Perimembranous VSD with Severe TR on Echocardiography

Submitted by varunbhb on Tue, 17/11/2009 - 10:07
in

 Perimembranous ventricular septal defect  (VSD) with severe Tricuspid regurgitation (TR).

3.5

Comments

Submitted by Beren (not verified) on Sat, 28/07/2012 - 13:15.

Usually an increase in LV-RV gadirent indicates that the VSD becomes more restrictive and it is a good sign. But a normal increase in gadirent is expected in the first few weeks of life due to fall in pulmonary vascular resistance which causes a decrease in pulmonary arterial and right ventricular systolic pressures.

Submitted by FeRs (not verified) on Thu, 26/07/2012 - 13:07.

I am a 41-yr. old CHD, ToF (tetralogy of fallot the clsuter of abnormalities described) survivor. This article mentions that other surgeries may be necessary. This is true for myself. My latter 20 years has brought a series of strokes, a second ToF repair that included a mechanical aortic valve and lastly, a repair of a discovered gap in the heart that was allowing unfiltered blood to pass into the blood stream. Repairs for all were available and I am finally whole' and ready for life to continue.

Submitted by varunbhb on Tue, 17/11/2009 - 10:10.

 Discussion: Mechanisms of tricuspid regurgitation in patient with perimembranous VSD:
1) Tricuspid valves clefts or dysplasia. 2) VSD jet pushing the tricuspid anterior leaflet to open tricuspid valves orifice. Mechanisms of spontaneous closure of membranous VSD: The tricuspid valve was responsible for most common mechanism of closure(85%) by either reduplication of leaflet tissue or adherence of septal leaflet to the margins of defect. Membranous VSD diminish in size or close spontaneously by aneurysm formation. So-call ventricular septal aneurysm is not a true aneurysm of the septaum membranaceum but rather a tissue out pouching from the tricuspid valve.Discussion: Mechanisms of tricuspid regurgitation in patient with perimembranous VSD:
1) Tricuspid valves clefts or dysplasia. 2) VSD jet pushing the tricuspid anterior leaflet to open tricuspid valves orifice. Mechanisms of spontaneous closure of membranous VSD: The tricuspid valve was responsible for most common mechanism of closure(85%) by either reduplication of leaflet tissue or adherence of septal leaflet to the margins of defect. Membranous VSD diminish in size or close spontaneously by aneurysm formation. So-call ventricular septal aneurysm is not a true aneurysm of the septaum membranaceum but rather a tissue out pouching from the tricuspid valve.

 

about                    contact us              disclaimer             faqs                    privacy           LifeHugger © 2012